CLASSIFICATION OF PERIODONTAL
DISEASES
The
recognition that diseases affecting the periodontal tissues can present
in different forms has been a major advance in our understanding of the
nature and prognosis of those disease in the last decade. As a result,
classification systems have been developed to reflect the variety of forms
of gingivitis and periodontitis. |
Consensus over the
exact nomenclature to be used has not been reached between different countries;
however, this information sheet provides an overview of how to classify
periodontal diseases, with the aim of assisting you in recording an appropriate
periodontal diagnosis on each patient's treatment record.
Gingivitis is an inflammatory
condition of the gingival tissues. Gingivitis lesions can be classified
as:
-
chronic marginal gingivitis
-
acute necrotizing ulcerative gingivitis (ANUG)
Chronic marginal gingivitis
is defined as inflammation of the marginal gingival tissues and is characterised
by redness, swelling and bleeding. It is essentially a reversible condition.
While gingivitis usually precedes the formation of periodontitis, this
is in no way an inevitable consequence of gingivitis.
Acute necrotizing ulcerative
gingivitis (ANUG) is an inflammatory destructive gingival condition
more commonly found in young adults, characterised by interproximal necrotic
ulcers covered by debris, painful to touch and readily bleeding. ANUG seems
to be related to poor plaque control with a pre- existing marginal gingivitis,
smoking and emotional stress.
Periodontitis is an
inflammatory disease of the periodontium characterized by the presence
of periodontal pockets and active bone resorption. Page and Schroeder (1982)
define five distinct forms of periodontitis:
-
prepubertal periodontitis
-
juvenile periodontitis
-
rapidly progressive periodontitis
-
adult periodontitis
-
acute necrotizing ulcerative
gingivo-periodonitis
The most common
type of these is the adult periodontitis. This is thought to commence
after the age of 30 years, and may affect any or all teeth, with either
vertical or horizontal patterns of bone loss. It rarely results in tooth
loss, although some patients do appear to suffer rapid destruction of periodontal
attachment at some stages. Acute exacerbations of the disease can superimpose
on the chronic form, with the formation of pus, which can lead to a periodontal
abscess. The majority of patients with adult periodontitis are relatively
straightforward cases to treat for the general dental practitioner. The
detection of other forms of periodontal diseases should be the responsibility
of the general dental practitioner; however referral to a periodontist
should be seriously considered for management of these conditions.
Prepubertal periodontitis
is an extremely rare condition, leading to early exfoliation of the deciduous
dentition, either with or without inflammation of the periodontal tissues.
It is usually associated with systemic illness, such as Papillon-Lefevre
Syndrome.
Juvenile periodontitis
has been well documented, and is characterised by severe periodontal breakdown
confined predominantly to the first permanent molars and/or incisors. The
onset of the disease is thought to coincide with puberty, when it is highly
active, but subsequently destruction may slow or spontaneously cease. The
prevalence of the disease is estimated to be about one per thousand children.
Clinically, the gingival tissues may appear completely normal, with very
little plaque present. Therefore, diagnosis relies heavily on the use of
a periodontal probe as a routine part of the dental examination and careful
examination of any radiographs taken. Juvenile periodontitis is usually
responsive to periodontal treatment; however, this may involve extensive
root-planing, antibiotic therapy and surgical access.
Rapidly progressive periodontitis
affects young
adults under the age of 35 years. It causes widespread destruction of the
periodontal tissues, affecting most teeth, and can result in early tooth
loss. The disease undergoes active and inactive phases. During the active
phase, the patient will often present with sore and swollen gingival tissues
that bleed profusely on gentle probing. Increased mobility of teeth may
also be noted. This active phase can last two to three weeks, before returning
to an inactive phase.
If the patient attends
during an inactive phase, there may be very few clinical cues to indicate
the extent of underlying periodontal destruction that has occurred. Again,
thorough periodontal probing and examination of radiographs is essential
to detect this condition. This form of periodontitis usually requires aggressive
treatment, with oral hygiene improvement, root-planing, antibiotic therapy
and periodontal surgery being likely components of treatment. Early detection
and referral for specialist care may give the patient the best prognosis.
The final category of periodontitis,
acute necrotizing ulcerative gingivo-periodontitis, is usually associated
with HIV-related periodontitis. It is often seen in the late stages of
HIV infection, when the patient is severely immune-compromised. It appears
to begin with typical signs of ANUG, but spreads rapidly to affect the
underlying periodontal tissues, causing necrosis of bone and rapid exfoliation
of teeth. Patients will often complain of a deepseated bone pain.
This brief overview
is a guide to forming a periodontal diagnosis for each of your patients.
For more detailed information, you may like to refer to:
1. Page RC, Schroeder
HE. Periodontitis in Man and other animals. Karger: Basel, 1982.
2. Williams DM, Hughes
FJ, Odell EW, Farthing PM. Pathology of periodontal disease. Oxford University
Press, 1992.
or phone the Dental Practice
Education Research Unit at the University of Adelaide on 008 80 5738.
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